Who will be the champion ? ARF, Avionics, BFTS, ERF or MTS?
All the below illnesses are known* to be caused by dichloromethane (DCM) also known as methylene chloride.
DCM was banned after a vote in the European Parliament in January 2009. The ban came into place in 2011 but the Irish Air Corps were still happy to let unsuspecting personnel use DCM without PPE or training in 2015 & likely beyond.
The Irish Air Corps finally issued PPE to all personnel using chemicals in 2017 a full 20 years after being told to do so by state body Forbairt.
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Klaassen CD, Ed. Casarett and Doull’s Toxicology: The Basic Science of Poisons, 6th Edition. New York: McGraw-Hill 2001. LaDou J, Ed. Occupational and Environmental Medicine, 3rd Edition. New York: Lange Medical/McGraw-Hill Company, 2004. Leikin JB, Davis A, Klodd DA, Thunder T, Kelafant GA, Paquette DL, Rothe MJ, Rubin R. Selected topics related to occupational exposures. Part V. Occupational cardiovascular disease. Disease-a-Month. 2000 Apr;46(4):311-322. Lynge E, Anttila A, Hemminki K. Organic solvents and cancer. Cancer Causes and Control. 1997 May;8(3):406-19. Rom WM. Environmental and Occupational Medicine, 3rd Edition. Philadelphia: Lippincott Williams & Wilkins Publishers, 1998. Severe Optic Neuropathy Caused by Dichloromethane Inhalation Atsushi Kobayashi, Akira Ando, Nobuko Tagami, Masahiko Kitagawa, Emi Kawai, Masako Akioka, Eiko Arai, Toshio Nakatani, Satoshi Nakano, Yoshie Matsui, and Miyo Matsumura Published Online:8 Dec 2008https://doi.org/10.1089/jop.2007.0100 Environmental and Chemical Toxins and Psychiatric Illness By James S. Brown. Publisher: American Psychiatric Association Publishing (27 Feb. 2002) Language: English ISBN-10: 0880489545
According to the World Health Organization (WHO), seven million people worldwide die as a consequence of air pollution every year. For around 20 years, studies have shown that air-borne particulate matter negatively affects human health. Now, in addition to already investigated particle sources like emissions from heating systems, industry and road traffic, aircraft turbine engine particle emissions have also become more important.
In a unique, innovative experiment, researchers have investigated the effect of exhaust particles from aircraft turbine engines on human lung cells.
The cells reacted most strongly to particles emitted during ground idling.
It was also shown that the cytotoxic effect is only to some extent comparable to that of particles from gasoline and diesel engines.The primary solid particles, i.e. those emitted directly from the source, have the strongest effect on people in its immediate vicinity.
Now a multidisciplinary team, led by lung researcher Marianne Geiser of the Institute of Anatomy at the University of Bern, together with colleagues from Empa Dübendorf and the University of Applied Sciences and Arts Northwestern Switzerland (FHNW), has shown that primary soot particles from kerosene combustion in aircraft turbine engines also cause direct damage to lung cells and can trigger an inflammatory reaction if the solid particles are inhaled in the direct vicinity of the engine.
The researchers demonstrated for the first time that the damaging effects also depend on the operating conditions of the turbine engine, the composition of the fuel, and the structure of the generated particles.
Beechcraft 200 Super King Air No 240 showing soiling of the engine panels with soot from exhaust gasses.
Extremely small particles in the nanoscale range
Particles emitted from aircraft turbine engines are generally ultrafine, i.e. smaller than 100 nm. By way of comparison, a human hair has a diameter of about 80,000 nm. When inhaled, these nanoparticles — like those from other combustion sources -efficiently deposit in the airways. In healthy people, the well-developed defense mechanisms in the lungs normally take care of rendering the deposited particles ineffective and removing them from the lungs as quickly as possible.
However, if the inhaled particles manage to overcome these defense mechanisms, due to their structure or physico-chemical properties, there is a danger for irreparable damage to the lung tissue. This process, already known to researchers from earlier experiments with particle emissions from gasoline and diesel engines, has now also been observed for particle emissions from aircraft engines.
Toxicity depends on the operating conditions of the turbines and the type of fuel
Evidence of increased cell membrane damage and oxidative stress in the cell cultures was identified. Oxidative stress accelerates ageing of cells and can be a trigger for cancer or immune system diseases.
Overall, according to the researchers, it has been demonstrated that the cell-damaging effect caused by exposure to particles generated by the combustion of gasoline, diesel and kerosene fuel are comparable for similar doses and exposure times.
Additionally, a similar pattern was found in the secretion of inflammatory cytokines after exposure to gasoline and kerosene fuel particles.
Aerosols: distance from the source is crucial
Aerosols are the finest solid or fluid substance suspended in the air. In combustion processes, the composition of ultrafine particles is highly variable. In addition, aerosols are unstable, and they are modified after their formation. Primary ultrafine solid particles have a high diffusion velocity. As a result, at high concentrations such particles either stick together or attach to other particles. Therefore, the effect of primary ultrafine particles depends on the distance from the source, implying that there is a difference depending on whether a person is close to the source (such as people at the roadside ) or at a greater distance (aircraft taxiing or taking off). Further research is needed to clarify how strong the impact would be at a greater distance from an aircraft engine
The layout of the Irish Air Corps base at Casement Aerodrome ensures that aircraft exhaust gasses are blown over populated sections of the airbase when winds are from the south, south east or south west. This includes hangars, offices, workshops and living in accommodation such as the apprentice hostel and married quarters. Calm weather also creates conditions where exhaust gasses linger in higher concentrations.
This results in all Irish Air Corps personnel (commissioned, enlisted, civilian & family) being exposed to emissions from idling aircraft engines, emissions that are known to cause harm.
In the mid 1990s a study of air pollution adjacent to the ramp area at Baldonnel was commissioned. This report relating to this study has gone missing.
Anecdotal evidence suggests increased prevalence of occupational asthma & adult onset asthma amongst serving & former personnel who served in Baldonnel or Gormanston aerodromes.
Older gas turbine engines produce dirtier exhaust gasses.
Idling gas turbine engines produce dirtier exhaust gasses.
Below are some of the gas turbine powered Air Corps aircraft that were powered by elderly engine designs.
Royal Australian Air Force (RAAF) personnel who worked with widely used jet fuel suffered damage to their body’s cells with unknown long-term consequences, according to groundbreaking research released after a Freedom of Information laws request.
Defence’s senior physician in occupational and environmental medicine, Dr Ian Gardner, described the findings as a “part of the puzzle” and a hypothesis-making study”, and pointed it out that it was one of a series of pieces of research currently underway.
“What it shows is there is evidence of small but persistent cellular damage,” Dr Gardner told the ABC. He said it was not yet clear what the long-term effects of that damage might be.
“For the future though there are a lot of other aircraft maintenance workers who have done similar jobs on other aircraft types, and now Defence and DVA and Air Force are considering what additional work should be done in relation to those other people who are not actually on the F-111 programs but have done essentially similar work,” Dr Gardner said.
The Jet Fuel Syndrome Study also shows that the fuel is more toxic to the body’s cells than the two solvents initially blamed for the sickness suffered by the deseal/reseal workers, and that the toxicity is even higher when those solvents and the fuel were mixed.
The results of the research project, headed by Professor Francis Bowling of Brisbane’s Mater Hospital, were handed to Defence last September, and have been the subject of significant scrutiny and review due to the potential significance of the findings.
They will give heart to former and serving Defence personnel who believe they have been left out in the cold by Defence after developing serious health complaints while working with fuel and other substances.
Junior Minister with responsibility for Defence said in the Dáil that he was assured by the Irish Air Corps that the RAAF F1-11 deseal/reseal exposure tragedy is completely different to any exposures at the Irish Air Corps.
Was the minister suggesting that Irish Air Corps gas turbine engines don’t run on jet fuel?
As the world frantically battles coronavirus, a leading Dutch neurologist warns of the next global pandemic — and this one, he says, is almost entirely of our own making.
Bastiaan Bloem, MD, a neurologist and professor at Radboud University Nijmegen Medical Center, says that over the next 20 years, the number of people with Parkinson’s disease (PD) will likely double — from the present 6.5 million to more than 13 million.
The main cause of this exponential jump: widespread exposure to herbicides, solvents, and other toxic chemicals used in agriculture and manufacturing.
“A pandemic, as everybody is now painfully aware, is a disease happening worldwide, to which no one is immune. PD fulfills all those criteria,” Bloem told Parkinson’s News Today in a phone interview from the Netherlands.
“Parkinson’s is now the fastest-growing neurological condition on the planet.”
Bloem, 53, points to the tight link between exposure to herbicides such as paraquat — a weed killer — and the risk of developing Parkinson’s.
“These chemicals were introduced worldwide after World War II, and many are still used today on our fields,” he said. “For this reason, farmers are at a markedly increased risk of developing Parkinson’s. If you feed a mouse paraquat — which is banned in China but not the U.S. — it will kill the dopamine-producing cells in the brain. These chemicals are tremendously toxic to the brain and have even been detected in milk, in supermarkets.”
Paraquat isn’t the only such chemical posing this risk. Trichloroethylene, a solvent used to clean metals and remove stains, has exactly the same effect on human brains. Yet it’s still widely used and is detectable in high concentrations in groundwater, he said.
“Parkinson’s is exploding in numbers, it’s a horribly debilitating disease, and it’s a costly disease that should matter to people and governments. We’re doing this to ourselves,” Bloem said. “But we can do something about it. We need to get rid of these toxic pesticides and move toward organic food. And we should take measures to protect people who work in these toxic environments.”
Trichloroethylene was used in Baldonnel for decades with ERF in particular receiving it in 220 litre drums. From ERF it was handed out without any precautions or training to anyone who asked for it. It was handed out in milk cartons, plastic coke bottles etc.
Trichloroethylene was used by all hangars & workshops in an ad-hoc basis usually with Trichloroethylene begged from ERF although some units did order it themselves. Personnel in the Air Corps museum also used Trike to help degrease parts & aircraft being restored for the museum.
Trichloroethylene was also used by both apprentices, tech & line personnel to carry out cleaning tasks in the Air Corps Training Depot while on training courses or during “war week”.
In at least 2 separate instances some floors in ACTD were completely destroyed by the use of Trichloroethylene being left overnight to clean them. In one incident Trichloroethylene dissolved through a traditional lino floor as far as the backing twine and in another incident few years later a tiled floor was destroyed after the tiles shriveled up & shrunk after Trichloroethylene was left overnight to clean a floor.
Trichloroethylene was also used by teenage apprentices to clean black marks off floors in the Apprentice Hostel and the Apprentice School.
At no point was anyone ever given training in the use of Trichloroethylene nor issued with appropriate PPE whilst working with the chemical.
A number of Irish Air Corps personnel have been diagnosed with early onset Parkinson’s disease
Concerns related to adverse health effects experienced by aircrew exposed to aircraft contaminated air have been ongoing for over 6 decades.
Unfiltered breathing air is supplied to the cabin via the engine compressor. The likelihood that oil leaking over the engine oil seals may enter the cabin air supply has prompted continuing debate about the hazards associated with exposure to neurotoxic substances and to the thermally degraded or pyrolysed mixture.
In this study, we undertook an in-depth investigation of aircrew involved in suspected aircraft contaminated air events.
Two studies were conducted to review the circumstances and symptoms of a cohort of aircrew working in the pressurized air environment of aircraft. A table of effects was then used for categorizing symptoms and reviewing other sources of data related to aircraft fluids and selected other conditions.
Both acute and chronic exposures to neurotoxic and a wide range of thermally degraded substances were confirmed, along with a clear pattern of acute and chronic adverse effects. The latter were supported by medical findings and diagnoses, notably involving the neurological, neurobehavioural and respiratory systems.
A clear cause and effect relationship has been identified linking the symptoms, diagnoses and findings to the occupational environment. Recognition of this new occupational disorder and a clear medical investigation protocol are urgently needed.
Download scientific research paper from the World Health Organisation…
We are curently updating our chemical listings. There were and are a very large number of chemical substances in use in the Irish Air Corps and amost all of these substances have ingredient chemicals.
For example, the corrosion inhibiter Mastinox 6856k, which many personnel used without any PPE, contains the below constituent chemicals. Among the ingredients include carcinogens, mutagens, immune sensitisers, irritants and just plain old toxic chemicals.
By following the links below you will arrive at tables showing the individual chemical names that we have extracted from some MSDS. Due to a recent Supreme Court ruling the list of chemicals is expected to grow. It is our intention to eventually create a database that links the MSDS to the individual constituents.
Bear the following in mind when using the table.
You can limit the number of entries you want to see
You can search for names such as “Tolouene” or “chromate”
You can sort by name, carcinogen, mutagen, reprotoxic etc.
Clicking on the name will take you to the Wikipedia page for that chemical
Clicking on the ECHA link for a chemical will take you to the European Chemicals Agency where you can look at the hazards in detail and view CAS number etc.
The tables do SCROLL sideways, the scroll bar is at the bottom of each table.
In 1995 the Irish Air Corps commissioned an independent third party organisation to carry out air quality testing in the Engine Repair Flight building. ERF comprised the Engine Overhaul facility, the Non Destructive Testing workshop and the Machine shop. Avionics Squadron was located in the same physical building as the ERF and the open attic spaces meant chemical vapours & fumes from either unit were free to flow in either direction.
The Avionics / ERF building was also less than 20m downwind from the 3m high Spray Paint Shop exhaust stack which exhausted benzene, hexamethylene diisocyanate, toluene and xylene.
Ambient Air Monitoring for Health & Safety at Work Report from the 2nd of August 1995 found that #Dichloromethane (also known as Methylene chloride) was found in some areas to be 175ppm. At the time The most stringent health and safety limits for #DCM were 50 ppm (TWA for 8 hours) and 126 ppm (STEL for 15 minutes).
Personnel, including other ranks employed in the Formation Safety Office, were never informed of these results. Avionics / ERF personnel were left in situ for a further 12 years before the workshops were finally condemned & demolished and needlessly exposed to #DCM and other chemicals.
Significantly Dichloromethane metabolises as Carbon Monoxide once inhaled but is lethal in many other different ways. We have at least 10 untimely deaths of men who worked in this building alone, their average age of death was 49.3 years and the youngest was only 32.
So we have one state body warning the general public about the dangers of Carbon Monoxide, while another state body hid evidence of a known carbon monoxide threat from personnel working in a heavily contaminated facility.
Samsung has apologised to employees who developed cancer at one of its computer chip manufacturing facilities following a ten-year legal battle.
The announcement comes after the company and a group representing ailing Samsung workers agreed to accept compensation terms and end a highly-publicised standoff. The company’s apology was part of the settlement.
Kim Ki-nam, the head of Samsung’s semiconductor business, said: “We sincerely apologise to the workers who suffered from illness and their families. We have failed to properly manage health risks at our semiconductor and LCD factories.”
Campaigners claim that 320 employees at Samsung have developed illnesses after being exposed to toxic chemicals at in its chip factories. They also claim that 118 people died as a result.
Here is a list of some of the chemicals used by Samsung and surprise, surprise all of them bar one were used by the Irish Air Corps in different hangars, labs & workshops at Baldonnel & Gormanston aerodromes.
In fact Trichloroethane was so “borrowed” by other units that almost every location at Baldonnel would send personnel up to the Engine Shop to obtain some TRIKE in plastic Coca Cola bottles, milk cartons, aerosol lids or any other vessel capable of begging some of the liquid. Trike was used to clean, degrease or even just remove black marks off floors.
This last usage meant that on at least 2 occasions floors in the Air Corps Training Depot were actually disolved in separate incidents years appart. One where old fashioned lino was dissolved back to the backing twine and another years later were a lecture room was mopped with a 25 litre drum of Trike that resulted in the vinyl floor tiles shrinking & curling up and the wall paint disolving & flowing off the walls onto the floor.
Used By Samsung
Used By Air Corps
aka TCE aka Trike
Thinners (containing Benzene, Toluene, Xylene).
Kim Ki-nam, the head of Samsung’s semiconductor business, said: "We sincerely apologise to the workers who suffered from illness and their families. We have failed to properly manage health risks at our semiconductor and LCD factories.”
You were not exposed to toxic chemicals.
If you were exposed to toxic chemicals you should have worn the PPE provided.
You should have relied upon the Chemical Training provided.
You should have used common sense.
Note that the “independent third party” investigator, Christopher O’Toole, is a retired barrister from the office of the Attorney General (an office incidentally being sued by exposed personnel..so much for third party independence). O’Toole could find no documentation to back up the Air Corps / State Claims Agency claim that PPE was provided nor that Chemical Training was provided….simply because it WASNT…not until 2017 a full 2 years after the whistleblower’s protected disclosures.
Furthermore O’Toole DID NOT investigate ILLNESS, O’Toole DID NOT investigate CHEMICAL EXPOSURE. O’Toole only really investigated whether documentation to prove Air Corps compliance with Health & Safety legislation existed prior to 2015 and he could find NONE.
My expertise is in the area of law and in carrying out this review it was my intention to examine compliance by the Air Corps with the relevant law and regulation. I was not in a position to consider the substances in use or any implications for human health arising from such use as these issues are outside my competence. The allegations concern both the current health and safety regime and compliance with that regime in a period stretching back over 20 years.
Consider the trappings of modern life: Calvin Klein Eternity, gasoline, Gore-Tex, hairspray, paint, particle board, polyurethane iPod cases.
Now imagine that you’re allergic to virtually all of them.
Environmentalists usually think about chemical toxicity as either a dramatic local crisis (Bhopal, Love Canal) or the simmering concern of those far away (breast-feeding mothers in the Arctic) or far in the future (our oft-evoked grandchildren). But for people suffering from Multiple Chemical Sensitivities, the chemical crisis is already here. Indeed, thanks to industrialisation, it is already everywhere. And, like so many environment-related health issues, it disproportionately affects the poor and, moreover, drives many once financially stable people into poverty.
As a disease, Multiple Chemical Sensitivities doesn’t have an official case definition yet (more on this soon), but rather refers to a broad range of adverse symptoms brought on by an even more broad array of everyday chemicals. These symptoms are often provoked at exposure levels far below those that seem to affect the rest of the population — levels virtually always present in our homes, workplaces, and social venues. They commonly include severe headaches, food intolerance, difficulty breathing, nausea, irritation of the eyes, ears, nose, throat, and skin, and disorientation or confusion, but there are many more.
Current theories range from a genetic predisposition to chemical injury, to neurological damage, to abnormalities in detoxifying enzymes, to a so-called “toxicant-induced loss of tolerance” to environmental stressors, in which one particular exposure to a toxic substance overwhelms a person’s system and leaves them unable to cope with exposures to a wide range of other toxins.
It can be even more difficult finding an MCS-safe job. Even if a workplace itself is a tolerable environment (rare, given the ubiquity of toxic building materials), basic job-related interactions with the general public can be impossible. “The way a typical story goes,” says Zwillinger, “is that people lose the ability to make a living because they can’t be out in the public arena” without getting ill. Some MCS patients find a way to work from home (assuming they’ve found safe housing) — but that option is seldom available to poorer Americans forced to rely on low-wage, low-skill jobs.
It’s a bitter irony, since many with MCS see themselves as canaries in the modern-day coal mine. As recently as 1986, the exquisitely sensitive yellow birds were used to detect the presence of dangerous gases in mine shafts, and when they showed signs of illness — when they ceased to sing — it was an unambiguous warning: evacuate.
As growing numbers of MCS sufferers are driven from their homes and jobs, pushed to the fringes of medical science and the brink of financial ruin, made sick by industrialised civilisation itself, we would do well to heed their equally urgent warning. And fast, because this time around we can’t evacuate. There’s nowhere else to go.
Air Corps Chemical Abuse Survivors are the canary in the coalmine for inflammatory illness being caused by ubiquitous chemical exposure in our home, work, vehicles, food & clothing.
The appalling high concentration chemical exposure suffered by Air Corps personnel caused chemical related illnesses in young fit men & women in a very short space of time making us a very valuable cluster for medicine & science to study.
Study us, fix us and there will be a public health dividend.
Epigenetics is the study of heritable phenotype changes that do not involve alterations in the DNA sequence. The Greek prefix epi- (ἐπι- “over, outside of, around”) in epigenetics implies features that are “on top of” or “in addition to” the traditional genetic basis for inheritance. Epigenetics most often denotes changes that affect gene activity and expression, but can also be used to describe any heritable phenotypic change. Such effects on cellular and physiological phenotypic traits may result from external or environmental factors, or be part of normal developmental program. The standard definition of epigenetics requires these alterations to be heritable, either in the progeny of cells or of organisms.
The term also refers to the changes themselves: functionally relevant changes to the genome that do not involve a change in the nucleotide sequence. Examples of mechanisms that produce such changes are DNA methylation and histone modification, each of which alters how genes are expressed without altering the underlying DNA sequence. Gene expression can be controlled through the action of repressor proteins that attach to silencer regions of the DNA.
These epigenetic changes may last through cell divisions for the duration of the cell’s life, and may also last for multiple generations even though they do not involve changes in the underlying DNA sequence of the organism; instead, non-genetic factors cause the organism’s genes to behave (or “express themselves”) differently.